Abstract
Osteonecrosis of the jaw (ONJ) presents clinically as exposed, necrotic bone in the maxilla or mandible of at least 8 weeks' duration, with or without the presence of pain, infection, or previous trauma in a patient who has not received radiation to the jaws.1-3 Although necrotic bone exposure has been reported in the jaws of a variety of patients not receiving bisphosphonates (BPs),4-9 the number of BP-related ONJ cases has continued to increase steadily since the first report in 2003.10 To date, a direct causal relationship between BP use and ONJ has not been established.
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