Abstract
Background: Orbital infarction is an unusual complication of a vaso-occlusive crisis (VOC) in sickle cell disease (SCD). The infarctive process produces an inflammatory response that can cause orbital compression syndrome. SCD is widely prevalent in Oman, providing a unique opportunity for identifying the influence of different ethnic backgrounds and sickle haplotypes in the etiopathogenesis of orbital infarction in SCD. Aim: To determine the role and significance of hematologic and genetic factors in Omani SCD patients with orbital infarction.
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