Abstract
Gestational diabetes (GDM) is traditionally thought to be a placenta endocrine disorder that carries short- and long-term cardiometabolic implications for the pregnant women and their offspring. Emerging evidence suggests that fetal sex has a direct impact on the development of GDM. We investigated the nutrient-sensing O-GlcNAc pathway as a potential mediator of sex-driven placenta dysfunction due to the unique location of the O-GlcNAc transferase (OGT) enzyme on the X chromosome.
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