Abstract
The attachment and migration of airway epithelial cells is an important aspect of the repair of antigen-induced inflammatory injury in patients with asthma. Cytoskeletal reorganization is required for the altered attachment and migration of epithelial cells. Vasodilator-stimulated phosphoprotein (VASP) mediates focal adhesion and actin filament binding in a variety of cells and acts as an inhibitor of cell movement. Phosphorylation of VASP via cyclic adenosine monophosphate-dependent and cyclic guanosine monophosphate-dependent protein kinases releases this “brake” on cell motility.
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