Abstract
Postoperative complications resulting from bariatric surgery can lead to severe vitamin-deficiency states. A patient who underwent bariatric bypass surgery and later developed Wernicke's encephalopathy prompted us to present her interesting case history for discussion. Although bariatric surgery is known to be a risk factor for Wernicke's encephalopathy, this diagnosis is only rarely evoked in the postoperative course. We recommend that the occurrence of digestive, psychiatric or neurological symptoms after bariatric surgery should suggest a thiamine deficiency that must be promptly assessed. Without waiting for the results, thiamine supplementation should be initiated.
Highlights
Support for the current epidemic of morbid obesity affecting industrialized countries requires a multidisciplinary approach in which surgery is a growing (Body Mass Index (B.M.I.) 40 or B.M.I. 35 and comorbidities).[1]
This type of treatment, which should be reserved for selected patients, allows for weight loss and improved cardiovascular satellites comorbidities of obesity such as diabetes, hypertension and dyslipidemia.[2]
The diagnosis of encephalopathy of Gayet-Wernicke is generally referred to the history of bariatric surgery: vertical gastroplasty size poses,[4] gastric ring,[5] and most often gastric bypass.[6]
Summary
Support for the current epidemic of morbid obesity affecting industrialized countries requires a multidisciplinary approach in which surgery is a growing (Body Mass Index (B.M.I.) 40 or B.M.I. 35 and comorbidities).[1]. Bariatric surgery appears to be another emerging condition that may lead to Wernicke’s encephalopathy.
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