Abstract
Directed transluminal atherectomy is a new catheter-mediated technique for the treatment of symptomatic peripheral and coronary artery atherosclerosis. 1 Unlike angioplasty, which may improve vessel patency by disrupting the plaque and stretching the arterial wall, the Simpson atherectomy catheter was specifically designed for percutaneous resection and removal of atherosclerotic plaques. 1–3 It was hypothesized that such an approach would allow for more predictable clinical results with fewer acute complications compared to balloon angioplasty. In the current study, the mechanism of directed atherectomy was investigated through experimental procedures performed on diseased segments of human arteries.
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