Abstract
Epidemiologic studies have repeatedly implicated infectious and noninfectious maternal immune activation (MIA) in the etiology of neuropsychiatric illnesses (1). MIA can trigger several pathophysiological processes, including inflammation and oxidative stress in maternal and fetal compartments, activation of maternal stress response systems, and temporary micronutrient and/or macronutrient deficiencies, as well as disruption of placental functions (2). When occurring during sensitive periods of fetal development, these pathophysiological processes have the potential to change the offspring’s neurodevelopmental trajectories and increase their risk to develop neuropsychiatric disorders later in life (2).
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
