Juvenile idiopathic arthritis presenting with rare extra-articular manifestations: A report of two cases

Case Reports1 February 1957CONSTRICTIVE PERICARDITIS AS SEQUEL TO HEMOPERICARDIUM: REPORT OF A CASE FOLLOWING ANTICOAGULANT THERAPYMAURICE H. LASZLO, M.D.MAURICE H. LASZLO, M.D.Search for more papers by this authorAuthor, Article, and Disclosure Informationhttps://doi.org/10.7326/0003-4819-46-2-403 SectionsAboutPDF ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinkedInRedditEmail ExcerptThe various causes of constrictive pericarditis have as yet not been thoroughly elucidated. Indeed, there is a wide divergence in various studies with respect to the incidence of the several known etiologic factors.1-4Undoubtedly tuberculous pericarditis is a fairly common precursor of constrictive pericarditis; pyogenic agents, polyserositis and previous nonspecific acute pericarditis are also at times implicated as causative factors. In addition, there still remains a large group of cases of indeterminate cause where the pathology of the pericardium, as found at surgery or autopsy, throws no light on the etiology. For example, no etiologic agent could be demonstrated in...Bibliography1. PaulCastlemanWhite OBPD: Chronic constrictive pericarditis: a study of 53 cases, Am. J. M. Sc. 216: 361, 1948. CrossrefMedlineGoogle Scholar2. AndrewsPickeringSellors GWGWTH: The aetiology of constrictive pericarditis with special reference to tuberculous pericarditis, together with a note on polyserositis, Quart. J. Med. 17: 291, 1948. MedlineGoogle Scholar3. ChamblissJaruszewskiBrofmanMartinFeil JREJBLJFH: Chronic cardiac compression (chronic constrictive pericarditis), Circulation 4: 816, 1951. CrossrefMedlineGoogle Scholar4. Moschcowitz E: Pathogenesis of constrictive pericardium, J. A. M. A. 153: 194, 1953. CrossrefMedlineGoogle Scholar5. Nichol ES: The risk of hemorrhage in anticoagulant therapy, Ann. West. Med. and Surg. 4: 71, 1950. MedlineGoogle Scholar6. GoldsteinWolff RL: Hemorrhagic pericarditis in acute myocardial infarction treated with bishydroxycoumarin, J. A. M. A. 146: 616, 1951. CrossrefMedlineGoogle Scholar7. IzzoStevensTomsykoskiRodriguez PARCAJCE: Hemopericardium associated with anticoagulant therapy, Arch. Int. Med. 92: 350, 1953. CrossrefGoogle Scholar8. WoodWolferthBellet FCCCS: Infarction of the lateral wall of the left ventricle: electrocardiographic characteristics, Am. Heart. J. 16: 387, 1938. CrossrefGoogle Scholar9. ThomsonFeil HWH: Infarction of the lateral wall of the left ventricle: pathologic and electrocardiographic study, Am. J. M. Sc. 207: 588, 1944. CrossrefGoogle Scholar10. Beck CS: The effect of surgical solution of chlorinated soda (Dakin's solution) in the pericardial cavity, Arch. Surg. 18: 1659, 1929. CrossrefGoogle Scholar11. DanielPuder GS: Pericarditis et pleuritis cholesterinea, Virchows Arch. f. path. Anat. 284: 853, 1932. CrossrefGoogle Scholar12. White PD: Heart disease, 1937, The Macmillan Company, New York. Google Scholar13. Beck CS: Acute and chronic compression of the heart, Am. Heart J. 14: 515, 1937. CrossrefGoogle Scholar14. Glenn EE: Traumatic constrictive pericarditis, J. Missouri M. A. 37: 7, 1940. Google Scholar15. Dejou L: Hemo-pericarde compressif, au second mais d'une plaie cardiaque mecounne, Mém. Acad. de chir. 72: 352, 1946. Google Scholar16. AdaJonesSheeran AEORAD: Partial pericardiectomy in a case of hemopericardium due to nonpenetrating trauma, J. Thoracic Surg. 20: 105, 1950. CrossrefGoogle Scholar17. BarkerJohnston PSFD: Chronic pericarditis with effusion, Circulation 2: 134, 1950. CrossrefMedlineGoogle Scholar18. McKusick VA: Chronic constrictive pericarditis. 1. Some clinical and laboratory observations, Bull. Johns Hopkins Hosp. 90: 3, 1952. MedlineGoogle Scholar19. Warburg E: Traumatic armour heart, Am. Heart J. 49: 633, 1955. CrossrefMedlineGoogle Scholar20. OverholtBurwellWoodburyWalker RHCSJWJH: Constrictive pericarditis and constrictive pleuritis treated by pericardiectomy and pulmonary decortication, J. Thoracic Surg. 23: 1, 1952. CrossrefGoogle Scholar21. ElkinCampbell DCRE: Cardiac tamponade: treatment by aspiration, Ann. Surg. 133: 623, 1951. CrossrefMedlineGoogle Scholar22. MaguireGage CHM: A discussion of cardiac tamponade: treatment by aspiration, by Elkin, D. C., and Campbell, R. E.21 Google Scholar23. EhrenhaftTaber JLRE: Hemopericardium and constrictive pericarditis, J. Thoracic Surg. 24: 355, 1952. CrossrefGoogle Scholar This content is PDF only. To continue reading please click on the PDF icon. Author, Article, and Disclosure InformationAffiliations: New York, N. Y.*Received for publication February 24, 1956.From the Medical Service of the United States Public Health Service Hospital, Memphis, Tennessee. The opinions expressed are those of the author and do not necessarily reflect those of the U. S. Public Health Service.Requests for reprints should be addressed to Maurice H. Laszlo, M. D., Montefiore Hospital, New York 67, N. Y. PreviousarticleNextarticle Advertisement FiguresReferencesRelatedDetails Metrics Cited byLeserbreif zum Artikel: Kardiogener Schock 4 Stunden nach akut-PTCA bei HinterwandinfarktEarly constrictive pericarditis and anemia after Dressler's syndrome and inferior wall myocardial infarction.Constrictive pericarditis after myocardial infarction.Constrictive pericarditis after myocardial infarction. Sequela of anticoagulant-induced hemopericardiumCardiac Tamponade Complicating the Postpericardiotomy SyndromeTraumatic Hemopericardium and Chronic Constrictive PericarditisConstrictive pericarditis associated with hemangioma of the pericardiumAnticoagulant-Induced Hemopericardium with TamponadeThe evolution and current concepts of the surgical treatment of constrictive pericarditisA case of traumatic pericarditis with chronic tamponade and constrictionManagement of pericarditis secondary to myocardial infarctionSmall bowel obstruction due to hemorrhage secondary to anticoagulant therapyPostpericardiotomy syndrome following penetrating stab wounds of the chest: Comparison with the postcommissurotomy syndromeHemorrhagic pericardial effusion following myocardial infarction associated with a ventricular aneurysmPostpericardiotomy Syndrome Following Surgery for Nonrheumatic Heart Disease 1 February 1957Volume 46, Issue 2Page: 403-413KeywordsAutopsy pathologyElectrocardiographyEtiologyHemorrhageLesionsMedical servicesPericardiumResearch laboratoriesSurgeryTuberculosis ePublished: 1 December 2008 Issue Published: 1 February 1957 PDF downloadLoading ...

Left Ventricular Function and Myocardial Contractility in Chronic Constrictive Pericarditis

Hematemesis From Varices in a 14-year-old Without Hepatobiliary Disease: A "Wandering" Diagnosis.

Evolving Consensus in Portal Hypertension Report of the Baveno IV Consensus Workshop on methodology of diagnosis and therapy in portal hypertension

Predicting Mortality in Systemic Sclerosis

Incidental esophageal varices

A case of refractory rheumatoid pericarditis

Background: The diagnosis of constrictive pericarditis requires a high degree of clinical suspicion due to non-specific sign and symptoms. In endemic area like Vietnam the presentation of constrictive tuberculous pericarditis is common. Here we report a case of severe calcific chronic constrictive tuberculous pericarditis. Case Report: A 25-year-old Vietnamese man presented with fatigue, progressive exertional dyspnea, ankle edema, puffiness of face, abdominal distention, non-productive cough, weight loss, night sweat, and evening fever. After physical examination and investigation, the patient was diagnosed as constrictive pericarditis. Histopathology of pericardial tissue removed via pericardiectomy confirmed the diagnosis of tuberculous pericarditis. Conclusion: A high index of suspicion for constrictive pericarditis consists of the association of signs and symptoms of right heart failure and impaired diastolic filling due to pericardial constriction. The mainstay of treatment of chronic constrictive tuberculous pericarditis is pericardiectomy and anti-tubercular therapy.

Nonselective Beta-Blockers in Compensated Cirrhosis: Preventing Variceal Hemorrhage or Preventing Decompensation?

SAT0263 Cohort Study of 80 Patients with Juvenile Idiopathic Arthritis during Transition from Pediatric To Adult Care

Multicenter timolol study: The value of timolol and hemodynamic monitoring in the management of portal hypertensive cirrhotics with no esophageal varices

Invited commentary

Objective To assess the predictive value of endoscopic ultrasonography (EUS) for esophageal varices (EV) bleeding by studying the relationship between esophageal collateral veins (ECV), portal vein (PV) trunk with its main branches and EV bleeding. Methods A retrospective cohort study of 114 cases of moderate and severe EV was conducted. The ECV level was determined through EUS. At the same time, diameters of PV, azygos vein (AIV) and spleen vein (SV) were measured through EUS. The predictive value of ECV level and diameters of PV, AIV, SV for EV bleeding were assessed during the 1-year follow-up, which started from the first EUS examination to EV bleeding or the end of follow-up. Results Single factor Cox regression analysis showed severe peri-ECV varices had higher risk than mild in EV bleeding(HR=4.081, 95%CI: 1.833-9.086, P=0.001); severe para-ECV varices had higher risk than mild in EV bleeding(HR=4.042, 95%CI: 1.814-9.005, P=0.001). Multivariable Cox retrospective analysis showed ECV level was an effective predictor for EV bleeding, when the peri-ECV and para-ECV were severe varices, EV bleeding risk increased to 3.831 3 (P=0.004 3) and 3.493 3 (P=0.003 1) times compared with mild respectively. Diameters of PV, AIV and SV could predict EV bleeding(PV AUC=0.959, P 13.65 mm (sensitivity=0.94, specificity=0.84), AIV>8.65 mm (sensitivity=0.94, specificity=0.89), SV>9.45 mm (sensitivity=0.90, specificity=0.67), EV bleeding risk increased significantly. Conclusion EUS is helpful to predict the risk of moderate and severe EV bleeding, and severe varices of ECV, PV, AIV, and SV can be used as indicators to predict risk of EV bleeding. Key words: Ultrasonography; Esophageal and gastric varices; Hemorrhage; Portal hypertension

Bleeding Stomal Varices: Case Series and Systematic Review of the Literature

Chronic constrictive pericarditis (CCP) is one of the common causes of cardiogenic cirrhosis; it is rare for a patient to have both CCP and recurrent black stool, so we consider that CCP causes cardiogenic cirrhosis. Cardiogenic cirrhosis caused portal hypertension which then resulted in gastrointestinal bleeding. Herein, we report a case of a 40-year-old Chinese woman suffering from CCP who had upper gastrointestinal bleed and had to undergo emergency surgery. Two years after the emergency surgery, multiple reexaminations showed significantly improved cardiac functions, hemoglobin, and WBC levels and gastrointestinal functions.