Abstract
The mechanisms of saphenous vein graft (SVG) in-stent restenosis (ISR) have not been well described. Intravascular ultrasound (IVUS) was used to evaluate 46 SVG ISR lesions (angiographic diameter stenosis >50% or IVUS minimal lumen area <4mm2) in 45 pts. Neoatherosclerosis was defined as either
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