Abstract
Purpose: Osteoarthritis (OA) is now recognized as an inflammatory condition and certain immunopathological phenomena have been implicated in the disease. One of the contributory factors is impaired gut permeability and translocation of bacterial components to the joints through circulation. Discovery of bacterial DNA in synovial tissue supported this assumption but its role in OA pathophysiology has not yet been elucidated. We aim to investigate immunostimulatory effects of human osteoarthritic chondrocytes in response to bacterial DNA.
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