Abstract

There is now ample evidence suggesting the involvement of prostaglandins (PGs) in some form of experimental ocular inflammation. Detectable amounts of PGs of the E and F types were found in aqueous humour of rabbit eyes injured either by paracentesis of the anterior chamber1,2, scratching the iris3 or non-perforating trauma4. The inflammatory response to such physical stimuli, such as blood aqueous barrier disruption, hyperaemia, miosis and intraocular pressure (IOP) elevation5,6, could be mimicked by local application or intracameral injection of PGs or arachidonic acid (AA)7–11.

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