Abstract
In the early 1960s, complete serial section studies through the whole thrombosed segment of occluded coronary arteries of myocardial infarction patients autopsied in St Louis, Mo, showed that all of the occluding thrombi had been caused by fissures of the surface of the underlying atherosclerotic plaques, fissures through which blood sometimes entered the plaque interior before they were sealed by the thrombi.1 Because essentially similar findings have since been made in Germany,2 Great Britain,3 Denmark,4 and Russia (A. Vichert, personal communication, 1985) and the same scenario was found to be responsible for cerebral artery thrombosis,5 it now seems certain that thrombosis in human atherosclerotic arteries is always triggered by a disruption …
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