Abstract
Background: Mutations in multiple genes have been implicated in familial atrial ibrillation (AF). KCNQ1 was the irst disease gene identiied for familial AF, however, some recent reports showed that potassium channel mutations are not a major cause of AF in the general population. We sought to determine the frequency of potassium channel gene mutations in patients with lone AF and characterized the electrophysiological properties of the detected mutation.
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