Abstract
Background and Aims : Glutamine (Gln) is a plasmatic amino acid and a strong inverse association has been observed between its plasma level and cardiometabolic traits. The glutaminase 2 (Gls2) in the liver is the main enzyme that metabolize glutamine into glutamate and some studies have recently revealed that SNPs in Gls2 were the major determinant of the plasma glutamine levels in humans. Altogether, these findings raise the question of the relevance of the hepatic glutaminolysis to the glutamine to glutamate ratio and the consequence it may have on atherosclerosis.
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