Abstract
The excretion of urinary amino acids, levels of plasma amino acids, and activity of renal ATPase were determined in control and experimentally induced magnesium deficient rats. Urinary amino acid ex-cretion increased significantly during the rogression of magnesium deficiency. This increase involved all amino acids studied. No significant changes were noted in plasma amino acid levels between control and deficient animals. Renal Ka-K ATPase was significantly greater in magnesium deficient rats than in control rats. This study suggests that the aminoaciduria seen in the magnesium deficient rat is a result of renal tubular dysfunction which may have as its basic etiology a disturbance of energy metabolism in renal tubular cells.
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Schedule a callMore From: Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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