Abstract
Since 1980 when L. Ludwig et al. [1] identified changes in the liver characteristic of alcohol-induced hepatitis in persons who didn’t misuse alcohol, intense study of non-alcoholic fatty liver disease (NAFLD) started. The definition of NAFLD has two major principles: a morphological pattern in biopsy ma terial microscopy completely corresponds to changes induced by alcoholic pathology and the major etiologic factors is lacking – alcoholic at hepatotoxic dosages. Obligatory condition is to rule out viral, drug, metabolic and other reasons [2].
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