Abstract
Introduction: The incidence of cholangiocarcinoma (CCA) is increasing worldwide and is associated with poor patient outcomes. Identification molecular features of CCA could be helpful in designing new therapeutic approaches. Cancer cells are often exposed to a metabolically challenging environment with scarce availability of nutrients. This metabolic stress leads to changes in the balance between endogenous synthesis and exogenous uptake of fatty acids (FAs), which are needed by cells to support their own growth.
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