Abstract
Introduction Alterations in consciousness and nonfocal abnormalities of the electroencephalogram in patients with supratentorial mass lesions are varied and inconstant, and appear to bear no direct relationship to the site of compression or the degree of raised intracranial pressure. Several previously anomalous features of space-occupying lesions above the tentorium have proved to be due to disordered function in the brain stem, which is distorted secondarily to transtentorial herniation of the temporal lobe. Meyer first described this herniation in 1920, but clinicopathological correlations were surprisingly slow to accumulate after this important publication. Kernohan and Woltman drew attention in 1929 to ipsilateral hemiplegia due to indentation of the crus cerebri by the tentorial edge. It was another 10 years before Reid and Cone (1939) showed experimentally that the dilated pupil associated with rapid cerebral compression (e.g., due to extradural hematoma) was related to the temporal hernia displacing the trunk of the third
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
