Abstract
Background: The beta-2 agonist terbutaline (terb) increases fetal heart rate (FHR) in fetuses with complete AV block (CAVB) due to either maternal Sjögren’s antibodies (SSA) or left atrial isomerism (LAI), but the electrophysiologic characteristics of the FHR augmentation have not been evaluated. We hypothesized that, reflecting the differing pathophysiology of LAI and SSA CAVB, the patterns of FHR acceleration in terb treated fetuses would differ between those with LAI and those with SSA.
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