Abstract
ConclusionsThe dose-response relationships which describe the effects of small amounts of vasopressin on femoral arterial pressure and flow have been presented. The IA and IV routes of administration were examined in 5 experiments on anesthetized dogs. The decrease in flow following IA administration and the secondary decrease in flow following IV administration have been attributed to vasoconstriction in the vascular bed supplied by the femoral artery. The primary increase in flow following IV administration has been discussed in terms of elevation of central arterial pressure and reflex vasomotor activity. It is postulated that sub-maximal antidiuretic amounts of exogenous vasopressin are vasoactive.
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More From: Proceedings of the Society for Experimental Biology and Medicine. Society for Experimental Biology and Medicine (New York, N.Y.)
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