Abstract
Abstract Shoot‐applied nickel (Ni) treatments produced symptomatology, foliar Ni accumulation, and cytological changes in tomato (Lycopersicon esculentum Mill.) similar to those caused by treatments with root‐applied nickel (Ni). Leaf damage resulting from 100 ug/ml foliar Ni‐treatments consisted of interveinal chlorosis and spotting necrosis which appeared histologically as tissue collapse, cell clumping, and chloroplast disintegration. Shoot‐treated plants accumulated more Ni in leaves than in roots; whereas the reverse was true in root‐treated plants. Interference with root‐to‐shoot manganese translocation was attributed to attenuated vascular tissue and phloem blockage. Evidence of reduced nutrient transport and inhibited meristem activity due to Ni toxicity presents a potential for crop damage from excessive Ni in the atmosphere as well as in the soil environment.
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Schedule a callMore From: Journal of Environmental Science and Health . Part A: Environmental Science and Engineering
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