Abstract
Ligation of the descending branch of the left coronary artery caused an increase in the permeability of the tissue-blood barriers of the myocardium in the ischemic zone and in the boundary zone for sulfacetamide sodium. Trasylol reduced the permeability of the myocardial tissue-blood barriers in these zones and did not affect permeability in the intact zone. The use of a fluorescein test revealed the presence of membranotropic effect of trasylol; the compound did not affect the osmotic resistance of the erythrocytesin vitro.
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