Abstract
The hallmark of liver injury is liver fibrosis. Ultimately, significant fibrosis deposition results in cirrhosis, whereby there are changes in liver architecture with nodule formation and ultimately, disturbances to liver function.1 Although the clinical features of liver decompensation are well described (ie, ascites, spider naevi, jaundice, signs of hepatic encephalopathy), patients who have early cirrhosis often have no clinical signs and might be entirely asymptomatic.2 The avoidance and prevention of liver fibrosis is one of the key objective for liver clinicians and is achieved through direct treatments such as antivirals, or through lifestyle modifications (eg, alcohol avoidance and weight loss).
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