Abstract
Plasmid-encoded copper (Cu2+) resistance in Escherichia coli was due to decreased uptake of Cu2+. The Cu2+-resistant E. coli Rtsl strain contained a 60 MDa plasmid which is known to encode for both Cu2+ and kanamycin resistance. A plasmid-free derivative of the same organism exhibited a greater uptake of Cu2+, and sensitivity to Cu2+ in both respiration and growth studies than the E. coli Rtsl strain.
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