Abstract
Polyoma virus has been extensively used to study degrees of cell differentiation (Georges et al. 1982; Tanaka et al. 1982) and tissue specificity (de Villiers et al. 1984; De Simone et al. 1985; Maione et al. 1985) by means of analyses of cell permissivity to viral growth. The viral nontranscribed regulatory region that directs early and late mRNA transcription, as well as DNA replication (Tyndall et al. 1981), has also been used as an enhancer for heterologous genes in chimeric plasmids (de Villiers and Shaffner 1981) to analyze the regulatory interactions of putative cell functions in different tissue cell lines (Khoury and Gruss 1983; Herbomel et al. 1984).
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