Abstract
Abrain arteriovenous malformation is characterised by rapid blood flow from arteries to veins due to the absence of an intervening capillary bed. The venous components develop arterialised walls and often become distended. “Nidus” is the term used to describe the tangled mass of abnormal arteriovenous connections. The nidus should be distinguished from the feeding vessels and draining veins. AVMs occur anywhere in the neuraxis but are much more frequent in the brain. Whilst AVMs can be discovered incidentally (15%) they most commonly present with a spontaneous intracranial haemorrhage (65%) or seizures (20%). 1
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