Abstract
It has been 50 years since Smith [1] and Weller et al. [2] separately reported the isolation and propagation of a cytopathogenic virus from tissues of infants with cytomegalic inclusion disease. In the intervening half century, much has been learned about congenital cytomegalovirus (CMV) infection, as well as the epidemiological, transmission, and molecular biological characteristics of CMV and the natural history of CMV infection. Unfortunately, no means of preventing congenital CMV infection or its cognitive, motor, or sensory sequelae is available or even on the visible horizon. In this issue of the
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