Abstract
The response of the mammalian kidney to changes in the composition of the urine seems to be mediated by intrarenal chemoreceptors. Two distinct populations of chemoreceptors have been identified in the rat kidney. One type responds to ischemia but not to changes in perfusion pressure;the other type is spontaneously active and is sensitive to the composition and concentration of intrarenal fluid. The author suggests that renal chemoreceptors may play a role in the development of experimental hypertension.
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