Abstract
Concentrations of immunoreactive TRH (TRH- ir ) in rat spinal cord were examined after traumatic injury. TRH- ir was significantly increased at the injury site (thoracic region) and rostral areas (cervical region), and significantly decreased below the injury site. Changes were delayed and time-dependent. Since TRH may serve as an excitatory neurotransmitter within the spinal cord, these changes may contribute to the functional alterations observed after spinal injury.
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