Abstract
Objectives: Previously, we found that treatment of mice with the inflammatory cytokine oncostatin M (OSM) improved cardiac function and suppressed remodeling after myocardial infarction. Since these results suggested a protective role of OSM under hypoxic conditions, we assessed whether activation of the OSM receptor (Oβ) directly affects survival of cardiomyocytes under hypoxia and alters morphology and expression of genes that are responsible for O2 handling and consumption.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
