Abstract
β-Adrenergic receptor blockade reduces many of the clinical manifestations of hyperthyroidism particularly tachycardia and dysrhythmias. β-Adrenergic receptor blocking drugs without intrinsic sympathomimetic activity are superior to those possessing it, probably because of the increased sensitivity to adrenergic receptor stimulation which is found in this condition. However, such treatment should not be considered as a long-term substitute for specific antithyroid therapy.
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