Abstract
The effects of acrylamide (ACR), a synthetic neurotoxic chemical compound on non-enzymatic and enzymatic stress markers and brain histopathology were studied in wistar rats. ACR (50 mg/300 ml) was ingested through drinking water on alternative days and brain tissues were collected on 13th and 27th days post ingestion for analysis. Results revealed that ACR causes significant increase in non-enzymatic stress markers like lipid peroxidation (p
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