Abstract
The burden of cardiovascular disease (CVD) worldwide remains high despite advances in diagnosis and management.1 Risk factor modification has become the cornerstone of CVD prevention. Because the burden of CVD remains high throughout the world, it is important to identify subclinical markers of CVD, in addition to traditional clinical risk factors, to aid in risk stratification and prognostication. Coronary artery calcification (CAC), as well as calcifications in extracoronary vascular beds, have provided great insights into the risk of developing clinical CVD.
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