Abstract
Background: The importance of central nervous system syndrome associated with neuronal surface antibodies has been increasing. A lot of studies suggested that disabling cerebellar ataxias may develop through immune-mediated mechanisms. We previously reported a new anti-neuronal antibody, anti-Sez6l2 antibody (Yaguchi et al. Journal of Neurology). Sez6l2 is highly expressed in the hippocampus and cerebellar cortex and is one of the type 1 membrane proteins.
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