Abstract
Background and Aims : ANGPTL3 and PCSK9 are known regulators of lipoprotein metabolism. Patients harboring homozygous loss of function mutations in the ANGPTL3 gene show reduced levels of circulating PCSK9, indicating a possible coordinate regulation of these two proteins. This study aimed to establish whether the two proteins can cross-regulate in different conditions of nutritional availability.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
