Abstract
<strong>Background:</strong> Camptocormia is defined as forward flexion of the spine that manifests during walking and standing and disappears in recumbent position. The various etiologies include idiopathic Parkinson’s disease, multiple system atrophy, myopathies, degenerative joint disease, and drugs. <strong>Case Report:</strong> A 67-year-old diabetic female presented with bradykinesia and camptocormia that started 1 year prior to presentation. Evaluation revealed levosulpiride, a dopamine receptor blocker commonly used for dyspepsia, to be the culprit. <strong>Discussion:</strong> It is well known that dopamine receptor blockers cause parkinsonism and tardive syndromes. We report a rare and unusual presentation of camptocormia attributed to this commonly used gastrointestinal drug in the Asian population.
Highlights
Camptocormia is defined as forward flexion of the spine (.45 ̊) that manifests during walking and standing and disappears in recumbent position.[1]
We report a patient who presented with camptocormia and evaluation disclosed an unusual cause
Camptocormia is defined as abnormal and severe flexion (.45 ̊) of the thoracolumbar spine that manifests during walking and standing and disappears in recumbent position.[1]. It can occur in sporadic Parkinson’s disease (PD), post-encephalitic and parkin-related PD as well as in multiple system atrophy.[2]
Summary
Sahil Mehta1*, Rajender Kumar2 & Vivek Lal[1 1] Department of Neurology, Post Graduate Institute of Medical Education and Research, Chandigarh, IN, 2 Department of Nuclear Medicine and PET/CT, Post Graduate Institute of Medical Education and Research, Chandigarh, IN
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