Abstract
Cardiac infiltration of amyloid fibril results in progressive cardiomyopathy with a grave prognosis and results in cardiac diseases such as congestive heart disease, cardiomyopathy, valvular heart disease, and arrhythmias. We present a rare case of cardiac amyloidosis initially misdiagnosed as syndrome X in which recurrent chest pain and progressive heart failure could be managed finally by heart transplantation.
Highlights
Amyloidosis is a clinical disorder caused by extracellular deposition of insoluble abnormal fibrils, derived from aggregation of misfolded normally soluble protein [1,2,3,4]
We present a rare case of cardiac amyloidosis initially misdiagnosed as syndrome X in which recurrent chest pain and progressive heart failure could be managed by heart transplantation
The increased left ventricular wall thickness in cardiac amyloidosis, is associated with low voltages on the ECG and this feature is a specific finding for cardiac amyloidosis [9,10]
Summary
Amyloidosis is a clinical disorder caused by extracellular deposition of insoluble abnormal fibrils, derived from aggregation of misfolded normally soluble protein [1,2,3,4]. Cardiac infiltration of amyloid fibril results in progressive cardiomyopathy with a grave prognosis and results in cardiac diseases such as congestive heart disease, cardiomyopathy, valvular heart disease, and arrhythmias [1,2,3,4]. A prominent clinical feature of cardiac amyloidosis is a syndrome of heart failure, characterized by restrictive hemodynamics and progressive deterioration of systolic function [1,2,3,4]. We reported an interesting case of cardiac amyloidosis initially misdiagnosed as syndrome X and we discussed the key findings of the disease along with the latest evidence regarding the management and prognosis of cardiac amyloidosis
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