Abstract
Left prefrontal cortex (PFC)-targeted transcranial magnetic stimulation (TMS) is an FDA-approved treatment for depression and a promising treatment for PTSD, two disorders characterized by heightened amygdala activity. PFC-targeted TMS can modulate amygdala functioning, but it’s unknown whether this modulation occurs via a causal pathway. Here we assess whether TMS-evoked cortical activity propagates to the amygdala via a cortical-subcortical white matter (WM) pathway, and whether WM properties influence the degree of signal propagation.
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