Abstract
Left prefrontal cortex (PFC)-targeted transcranial magnetic stimulation (TMS) is an FDA-approved treatment for depression and a promising treatment for PTSD, two disorders characterized by heightened amygdala activity. PFC-targeted TMS can modulate amygdala functioning, but it’s unknown whether this modulation occurs via a causal pathway. Here we assess whether TMS-evoked cortical activity propagates to the amygdala via a cortical-subcortical white matter (WM) pathway, and whether WM properties influence the degree of signal propagation.
Sign-in/Register to access full text options 
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
