Abstract
The rate of mortality from acute respiratory distress syndrome (ARDS) has reportedly reached as high as 50–75%.1−3 The risk of ARDS development increases after severe blunt thoracic trauma (BTT) because of a higher likelihood for lung contusion4 and acute depression of cardiac function.5, 6 Monitoring of oxygen transport in patients with ARDS has shown that oxygen delivery and consumption were significantly higher in the survivors compared to nonsurvivors.7 This suggests that maintenance of oxygen delivery at optimal levels can potentially enable the reversal of ARDS.8 In cases of severe BTT, these oxygen transport variables may be induced by early cardiorespiratory dysfunction6, 9 which requires inotropic support.6, 8, 10 On the strength of these data, it is reasonable to conclude that the prevention and correction of oxygen deficiency are basic to intensive care during ARDS.
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