Abstract
Phosphatidyinositol-4,5-bisphosphate (PIP2) has an obligatory role in activating heteromeric TRPC1 subunit-containing channels in vascular myocytes, which also requires protein kinase C (PKC)-mediated phosphorylation of TRPC1 proteins. The aim of the present work was to further investigate these proposed activation mechanisms in freshly isolated mesenteric artery myocytes from TRPC1-/- mice.
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