Abstract
Acid amyloglucosidase activity in human fetal liver was found to display latency. Increase of ‘<i>free</i>’ enzyme activity was elicited <i>in vitro</i> by lowering of homogenate osmolarity or addition of detergent. A moderate but highly significant rise in the ‘<i>free</i>’ enzyme activity of the liver was found after in vivo exposition to 10 min of asphyxia. It is suggested that the increase of hepatic ‘<i>free</i>’ acid amyloglucosidase activity during acute asphyxia might be of significant relevance for the knowledge of the mechanisms behind the accompanying rise of plasma glucose and hepatic glucose production.
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