Acetylation regulates monopolar attachment at multiple levels during meiosis I in fission yeast.

Abstract

Corrigendum3 July 2017free access Acetylation regulates monopolar attachment at multiple levels during meiosis I in fission yeast Ayano Kagami Ayano Kagami Search for more papers by this author Takeshi Sakuno Takeshi Sakuno Search for more papers by this author Yuya Yamagishi Yuya Yamagishi Search for more papers by this author Tadashi Ishiguro Tadashi Ishiguro Search for more papers by this author Tatsuya Tsukahara Tatsuya Tsukahara Search for more papers by this author Katsuhiko Shirahige Katsuhiko Shirahige Search for more papers by this author Koichi Tanaka Koichi Tanaka Search for more papers by this author Yoshinori Watanabe Yoshinori Watanabe Search for more papers by this author Ayano Kagami Ayano Kagami Search for more papers by this author Takeshi Sakuno Takeshi Sakuno Search for more papers by this author Yuya Yamagishi Yuya Yamagishi Search for more papers by this author Tadashi Ishiguro Tadashi Ishiguro Search for more papers by this author Tatsuya Tsukahara Tatsuya Tsukahara Search for more papers by this author Katsuhiko Shirahige Katsuhiko Shirahige Search for more papers by this author Koichi Tanaka Koichi Tanaka Search for more papers by this author Yoshinori Watanabe Yoshinori Watanabe Search for more papers by this author Author Information Ayano Kagami, Takeshi Sakuno, Yuya Yamagishi, Tadashi Ishiguro, Tatsuya Tsukahara, Katsuhiko Shirahige, Koichi Tanaka and Yoshinori Watanabe EMBO Reports (2017)18:1263-1263https://doi.org/10.15252/embr.201744401 This article corrects the following: Acetylation regulates monopolar attachment at multiple levels during meiosis I in fission yeast07 October 2011 ToolsAdd to favoritesDownload CitationsTrack CitationsPermissions ShareFacebookTwitterLinked InMendeleyWechatReddit Figures & Info In Figure 2A, due to over-contrasting of the top two Western blot panels (AcPsm3), there is a loss of signal, in particular in the top right panel. Further, the right two panels were derived from the same blot, which was stripped and reprobed. The left two panels were derived from different blots of the same sample. This limits the quantitative information content of the published figure, but it does not change the conclusions derived from the experiment. In the corrected figure, the two left panels are now assembled from the same blot that was stripped and reprobed, and the contrast is properly adjusted in all panels. All these blots were processed in parallel in the course of the same experiment. Previous ArticleNext Article Read MoreAbout the coverClose modalView large imageVolume 18,Issue 7,July 2017Cover: In this issue, Fernandez‐Alonso et al report a molecular switch involving BET bromodomain proteins, which controls Embryonic Stem Cell (ESC) differentiation. BET family members drive distinct patterns of signalling and gene expression, such that interchange between BET proteins coordinates pluripotent exit with differentiation to mesendoderm and pancreatic/hepatic progenitor cells. The image shows mouse ESCs stained for Nanog protein (red), Dnmt3b protein (green) and DNA (blue). From Rosalia Fernandez‐Alonso, Greg M Findlay and colleagues: Brd4–Brd2 isoform switching coordinates pluripotent exit and Smad2‐dependent lineage specification. For detail, see Article on page 1108. Scientific images by Francisco Bustos, MRC Protein Phosphorylation and Ubiquitylation Unit, University of Dundee. Volume 18Issue 71 July 2017In this issue RelatedDetailsLoading ...