Abstract
Many researchers reported rates of infected dogs with T. canis and cats with Toxocara cati all over the world. The degree of tissue damage in the host and the concomitant elicitation of signs and symptoms are varied in different invaded tissues. The liver, lungs and central nervous system, including the eyes, are considered the most sensitive organs. In addition, the number of migrating juveniles and the age of the host. Inflammation manifests as eosinophilic granulomas. The immediate hypersensitivity responses to dying and dead larvae in the viscera, including the lungs, liver and brain, produce symptoms characteristic of VLM. The current review discusses the Toxocara sp. infection from the historical background, taxonomy, lifecycle, pathogeneses, clinical signs, epidemiology, diagnosis, control and treatment. And provides an overview of existing literature and data on the Toxocaraiasis with their references.
Highlights
In 1952, [3] reported similar cases in a series of children suffered high circulating eosinophilia and severe, long-term, multisystem disease. From this group of patients, they described most of the clinical features of VLM and in histopathological sections of tissues obtained at biopsy, correctly classified the causative agents as the larva of either T. canis or T. cati
The disease manifested with fever; enlargement and necrosis of the liver [19]; enlargement of the spleen; lower respiratory symptoms; eosinophilia sometimes raised up to 70% [20] and hypergammaglobulinemia of immunoglobulin M (IgM), IgG, and IgE classes
During the last two decades, cutaneous manifestations such as chronic urticaria, chronic pruritus and miscellaneous eczema, in patients with Toxocara antibodies, have been studied by different investigators. These cutaneous manifestations were the only signs indicating the presence of the disease and they were cured after antihelmintic treatment when there was good patient compliance
Summary
Human infection with Toxocariasis was first described by Wilder [2]. The latter he identified a nematode larva of unknown species within a retinal granuloma of a child. In 1952, [3] reported similar cases in a series of children suffered high circulating eosinophilia and severe, long-term, multisystem disease. From this group of patients, they described most of the clinical features of VLM and in histopathological sections of tissues obtained at biopsy, correctly classified the causative agents as the larva of either T. canis or T. cati. Small numbers of reactivated larvae may be shed in the milk
Sign-in/Register to view highlights & summary Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
