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Abstract
Quorum sensing is a cell-cell communication mechanism widely employed by bacteria to control various biological functions and pathogenicity. In this study, we demonstrated that Enterobacter cloacae employs indole-3-ethanol as a quorum-sensing signal to control biological functions and virulence. We also revealed that indole-3-ethanol from E. cloacae effectively inhibits biofilm formation and virulence in Shigella sonnei. Our findings not only suggest the important role of indole-3-ethanol in the regulation of the pathogenicity of E. cloacae but also provide new insights into the development of indole-3-ethanol as an anti-virulence agent against S. sonnei.
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