Abstract
Acute respiratory distress syndrome (ARDS) is the pulmonary manifestation of a systemic inflammatory process that is triggered by various events. Because of the various cytokines and vasoactive peptides secreted by the damaged endothelium, the vascular permeability is increased and this results in the migration of neutrophils, eosinophils, macrophages and other inflammatory cells, and also the leakage of protein-rich fluid. Edema consequently develops in the alveolus, and respiratory distress and hypoxia are then induced. Various attempts have recently been reported as conservative treatments for ARDS, but most of these treatments are at the experimental stage, and the mortality still reaches up to 40-60% 1-3) .
Talk to us
Join us for a 30 min session where you can share your feedback and ask us any queries you have
Schedule a callDisclaimer: All third-party content on this website/platform is and will remain the property of their respective owners and is provided on "as is" basis without any warranties, express or implied. Use of third-party content does not indicate any affiliation, sponsorship with or endorsement by them. Any references to third-party content is to identify the corresponding services and shall be considered fair use under The CopyrightLaw.
