Abstract
Guillain-Barré syndrome (GBS) is an acute inflammatory polyradiculoneuropathy that is hypothesized to be most commonly caused through the autoimmune mechanism of molecular mimicry. Typical GBS symptoms include back pain, paresthesiae in the limbs, and ascending, symmetrical muscle weakness. Examination findings in GBS include muscle weakness, areflexia, and a usually mild sensory loss. The majority of GBS patients report an infection one to four weeks before symptom onset. Zika virus (ZIKV) has now been associated with GBS in multiple case-control studies. GBS patients must be monitored for signs of impending respiratory failure and autonomic dysfunction. The treatment for ZIKV-associated GBS is the same as with other GBS cases; intravenous immunoglobulin and plasmapheresis are both thought to be equally effective treatments in ameliorating the condition. The prognosis for recovery from ZIKV-associated GBS is very good, based on the previously reported case series.
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