Abstract
Neuronal nitric oxide synthase (NOS-I) and its adaptor protein (NOS1AP) have repeatedly been associated with schizophrenia. NOS1AP competes with interaction between NOS-I and PSD-95, which has been hypothesized to contribute to schizophrenia-related phenotypes. Here we aimed to investigate the relevance of NOS-I/PSD-95-interaction in schizophrenia-related behaviors by viral overexpression of proteins disrupting this interaction.
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