Abstract
Skin diseases affect 900 million people worldwide. Skin colonization by Staphylococcus aureus ( S. aureus) is a hallmark of Atopic Dermatitis (AD), the most common inflammatory skin disease (ISD). The expression of antimicrobial peptides (AMPs) such as S100A8 (A8), S100A9 (A9) and the A8/A9 heterodimer Calprotectin (CP) was reported to be elevated in ISDs, which likely influences S. aureus growth. In the JunBΔep genetic mouse model of AD, constitutive epidermal deletion of JunB leads to skin inflammation with S.
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