Abstract
Hidradenitis suppurativa (HS) is a chronic, inflammatory skin disease of the apocrine gland-rich (AGR) skin regions with a well characterized Th1/17 cytokine milieu. According to current literature data, keratinocytes are crucial drivers of the first pathogenic steps of HS. However, the possible role of permeability barrier alteration, as KC activating event, has not been studied in detailed in HS. Our main focus was to study the permeability barrier of non-lesional HS (HS NL) and lesional HS (HS L) skin and compare them to that of the healthy AGR region.
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