Abstract
The present expeiments examined the influence of anoxia on spontaneous discharges of cochlear single units in guinea pigs; in several animals the combined effect of anoxia and sodium salicylate was observed. Anoxia was induced by stopping the respirator. Following the induction of anoxia, there was an initial increase in the number of spontaneous discharges in 19 of the 34 cochlear single units tested, after the amplitude of the discharges gradually decreased. The remaining 15 neurons displayed no increase during anoxia, although the same final disappearance of discharges was observed. Neurons showing high frequencies (over 100sp/sec) during normal respiration showed increase during anoxia. In 5 animals pretreated with sodium salicylate, a remarkable increase in the spike rates of spontaneous discharges were noted following the induction of anoxia.
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